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AF1q is a universal marker of neuroblastoma that sustains N-Myc expression and drives tumorigenesis.

  • Read more about AF1q is a universal marker of neuroblastoma that sustains N-Myc expression and drives tumorigenesis.

Combined BET bromodomain and CDK2 inhibition in MYC-driven medulloblastoma.

  • Read more about Combined BET bromodomain and CDK2 inhibition in MYC-driven medulloblastoma.

Epidermal growth factor receptor and EGFRvIII in glioblastoma: signaling pathways and targeted therapies.

  • Read more about Epidermal growth factor receptor and EGFRvIII in glioblastoma: signaling pathways and targeted therapies.

Inhibition of WNT signaling attenuates self-renewal of SHH-subgroup medulloblastoma.

  • Read more about Inhibition of WNT signaling attenuates self-renewal of SHH-subgroup medulloblastoma.

RasGRP1 overexpression in T-ALL increases basal nucleotide exchange on Ras rendering the Ras/PI3K/Akt pathway responsive to protumorigenic cytokines.

  • Read more about RasGRP1 overexpression in T-ALL increases basal nucleotide exchange on Ras rendering the Ras/PI3K/Akt pathway responsive to protumorigenic cytokines.

Alternative splicing in cancer: implications for biology and therapy.

  • Read more about Alternative splicing in cancer: implications for biology and therapy.

Upregulation of the microRNA cluster at the Dlk1-Dio3 locus in lung adenocarcinoma.

  • Read more about Upregulation of the microRNA cluster at the Dlk1-Dio3 locus in lung adenocarcinoma.

Myc proteins as therapeutic targets.

  • Read more about Myc proteins as therapeutic targets.

Cyclic GMP-dependent protein kinase II inhibits cell proliferation, Sox9 expression and Akt phosphorylation in human glioma cell lines.

  • Read more about Cyclic GMP-dependent protein kinase II inhibits cell proliferation, Sox9 expression and Akt phosphorylation in human glioma cell lines.

RNA interference against a glioma-derived allele of EGFR induces blockade at G2M.

  • Read more about RNA interference against a glioma-derived allele of EGFR induces blockade at G2M.

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