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Neonatal stroke enhances interaction of microglia-derived extracellular vesicles with microglial cells.

  • Read more about Neonatal stroke enhances interaction of microglia-derived extracellular vesicles with microglial cells.

Neuroprotective effects of a dendrimer-based glutamate carboxypeptidase inhibitor on superoxide dismutase transgenic mice after neonatal hypoxic-ischemic brain injury.

  • Read more about Neuroprotective effects of a dendrimer-based glutamate carboxypeptidase inhibitor on superoxide dismutase transgenic mice after neonatal hypoxic-ischemic brain injury.

21(st) Century Research in Child Neurology.

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Delayed erythropoietin therapy improves histological and behavioral outcomes after transient neonatal stroke.

  • Read more about Delayed erythropoietin therapy improves histological and behavioral outcomes after transient neonatal stroke.

MiR-139-5p inhibits HGTD-P and regulates neuronal apoptosis induced by hypoxia-ischemia in neonatal rats.

  • Read more about MiR-139-5p inhibits HGTD-P and regulates neuronal apoptosis induced by hypoxia-ischemia in neonatal rats.

Enhanced NMDA receptor tyrosine phosphorylation and increased brain injury following neonatal hypoxia-ischemia in mice with neuronal Fyn overexpression.

  • Read more about Enhanced NMDA receptor tyrosine phosphorylation and increased brain injury following neonatal hypoxia-ischemia in mice with neuronal Fyn overexpression.

Cerebellar abnormalities following hypoxia alone compared to hypoxic-ischemic forebrain injury in the developing rat brain.

  • Read more about Cerebellar abnormalities following hypoxia alone compared to hypoxic-ischemic forebrain injury in the developing rat brain.

Erythropoietin promotes hippocampal neurogenesis in in vitro models of neonatal stroke.

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Osteopontin is extensively expressed by macrophages following CNS demyelination but has a redundant role in remyelination.

  • Read more about Osteopontin is extensively expressed by macrophages following CNS demyelination but has a redundant role in remyelination.

Neonatal mice lacking functional Fas death receptors are resistant to hypoxic-ischemic brain injury.

  • Read more about Neonatal mice lacking functional Fas death receptors are resistant to hypoxic-ischemic brain injury.

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