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UCSF School of Medicine | Department of Pediatrics UCSF Medical Center

Heme oxygenase-2 modulates early pathogenesis after traumatic injury to the immature brain.

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HIF-1 alpha-deficient mice have increased brain injury after neonatal hypoxia-ischemia.

  • Read more about HIF-1 alpha-deficient mice have increased brain injury after neonatal hypoxia-ischemia.

Does inflammation after stroke affect the developing brain differently than adult brain?

  • Read more about Does inflammation after stroke affect the developing brain differently than adult brain?

Erythropoietin sustains cognitive function and brain volume after neonatal stroke.

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Individual differences in behavioral, physiological, and genetic sensitivities to contexts: implications for development and adaptation.

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Third pathophysiology of prenatal cocaine exposure.

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Antioxidant status alters levels of Fas-associated death domain-like IL-1B-converting enzyme inhibitory protein following neonatal hypoxia-ischemia.

  • Read more about Antioxidant status alters levels of Fas-associated death domain-like IL-1B-converting enzyme inhibitory protein following neonatal hypoxia-ischemia.

Erythropoietin enhances long-term neuroprotection and neurogenesis in neonatal stroke.

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Traumatic injury to the immature brain results in progressive neuronal loss, hyperactivity and delayed cognitive impairments.

  • Read more about Traumatic injury to the immature brain results in progressive neuronal loss, hyperactivity and delayed cognitive impairments.

Overexpression of glutathione peroxidase protects immature murine neurons from oxidative stress.

  • Read more about Overexpression of glutathione peroxidase protects immature murine neurons from oxidative stress.

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