Skip to main content
University of California San Francisco
UCSF School of Medicine | Department of Pediatrics UCSF Medical Center

Elevated End-Tidal Carbon Monoxide Concentration in Children with Sickle Cell Anemia.

  • Read more about Elevated End-Tidal Carbon Monoxide Concentration in Children with Sickle Cell Anemia.

Dietary Intake Insufficient to Support Nutritional Adequacy in Patients with Thalassemia.

  • Read more about Dietary Intake Insufficient to Support Nutritional Adequacy in Patients with Thalassemia.

Subclonal Mutations in SETBP1 Predict Relapse in Juvenile Myelomonocytic Leukemia.

  • Read more about Subclonal Mutations in SETBP1 Predict Relapse in Juvenile Myelomonocytic Leukemia.

The CXCR4/CXCL12 Axis Mediates Chemotaxis, Survival, and Chemoresistance in T-Cell Acute Lymphoblastic Leukemia.

  • Read more about The CXCR4/CXCL12 Axis Mediates Chemotaxis, Survival, and Chemoresistance in T-Cell Acute Lymphoblastic Leukemia.

MAPK Signaling Cascades Mediate Distinct Glucocorticoid Resistance Mechanisms in Pediatric B-Precursor ALL.

  • Read more about MAPK Signaling Cascades Mediate Distinct Glucocorticoid Resistance Mechanisms in Pediatric B-Precursor ALL.

Abstract 17814: Prenatal Diagnosis is Associated with a Lower Rate of Pre-Operative Brain Injury in Newborns with Transposition of the Great Arteries.

  • Read more about Abstract 17814: Prenatal Diagnosis is Associated with a Lower Rate of Pre-Operative Brain Injury in Newborns with Transposition of the Great Arteries.

Differential expression of the nuclear-encoded mitochondrial transcriptome in pediatric septic shock.

  • Read more about Differential expression of the nuclear-encoded mitochondrial transcriptome in pediatric septic shock.

Gene therapy for haemophilia.

  • Read more about Gene therapy for haemophilia.

Corticosteroids and pediatric septic shock outcomes: a risk stratified analysis.

  • Read more about Corticosteroids and pediatric septic shock outcomes: a risk stratified analysis.

138 Iron Overload and Hemolysis Modulate Monocytes and Inflammation in ß-Thalassemia.

  • Read more about 138 Iron Overload and Hemolysis Modulate Monocytes and Inflammation in ß-Thalassemia.

Pagination

  • Previous page ‹‹
  • Page 127
  • Next page ››
Subscribe to

Pediatrics Home

MAKE A GIFT

© 2026 The Regents of the University of California. The University of California San Francisco  |  UCSF Department of Pediatrics                                                                                   Accessibility  Privacy Policy  Terms of Use  A-Z Website List