Skip to main content
University of California San Francisco
UCSF School of Medicine | Department of Pediatrics UCSF Medical Center

miR-24 inhibits apoptosis and represses Bim in mouse cardiomyocytes.

  • Read more about miR-24 inhibits apoptosis and represses Bim in mouse cardiomyocytes.

Gain of MYC underlies recurrent trisomy of the MYC chromosome in acute promyelocytic leukemia.

  • Read more about Gain of MYC underlies recurrent trisomy of the MYC chromosome in acute promyelocytic leukemia.

Expansion of immunoglobulin-secreting cells and defects in B cell tolerance in Rag-dependent immunodeficiency.

  • Read more about Expansion of immunoglobulin-secreting cells and defects in B cell tolerance in Rag-dependent immunodeficiency.

Cytosolic PLA2 is required for CTL-mediated immunopathology of celiac disease via NKG2D and IL-15.

  • Read more about Cytosolic PLA2 is required for CTL-mediated immunopathology of celiac disease via NKG2D and IL-15.

Complementation of a pathogenic IFNGR2 misfolding mutation with modifiers of N-glycosylation.

  • Read more about Complementation of a pathogenic IFNGR2 misfolding mutation with modifiers of N-glycosylation.

A viral CTL escape mutation leading to immunoglobulin-like transcript 4-mediated functional inhibition of myelomonocytic cells.

  • Read more about A viral CTL escape mutation leading to immunoglobulin-like transcript 4-mediated functional inhibition of myelomonocytic cells.

Regulation of AID expression in the immune response.

  • Read more about Regulation of AID expression in the immune response.

Reprogramming of CTLs into natural killer-like cells in celiac disease.

  • Read more about Reprogramming of CTLs into natural killer-like cells in celiac disease.

Granulocyte/macrophage colony-stimulating factor and accessory cells modulate radioprotection by purified hematopoietic cells.

  • Read more about Granulocyte/macrophage colony-stimulating factor and accessory cells modulate radioprotection by purified hematopoietic cells.

Immune selection for altered antigen processing leads to cytotoxic T lymphocyte escape in chronic HIV-1 infection.

  • Read more about Immune selection for altered antigen processing leads to cytotoxic T lymphocyte escape in chronic HIV-1 infection.

Pagination

  • Previous page ‹‹
  • Page 3
  • Next page ››
Subscribe to The Journal of experimental medicine

Pediatrics Home

MAKE A GIFT

© 2026 The Regents of the University of California. The University of California San Francisco  |  UCSF Department of Pediatrics                                                                                   Accessibility  Privacy Policy  Terms of Use  A-Z Website List