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Incidence and outcomes of treatment-associated hepatotoxicity during pediatric acute lymphoblastic leukemia induction therapy: a Reducing Ethnic Disparities in Acute Leukemia (REDIAL) Consortium report.

  • Read more about Incidence and outcomes of treatment-associated hepatotoxicity during pediatric acute lymphoblastic leukemia induction therapy: a Reducing Ethnic Disparities in Acute Leukemia (REDIAL) Consortium report.

Early cytokine and chemokine signals shape the anti-AML activity of bispecific engager-secreting T cells.

  • Read more about Early cytokine and chemokine signals shape the anti-AML activity of bispecific engager-secreting T cells.

A transgenic mouse model of Down syndrome acute lymphoblastic leukemia identifies targetable vulnerabilities.

  • Read more about A transgenic mouse model of Down syndrome acute lymphoblastic leukemia identifies targetable vulnerabilities.

LNK/SH2B3 as a novel driver in juvenile myelomonocytic leukemia.

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Down syndrome and leukemia: from basic mechanisms to clinical advances.

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Prognostic impact of pretreatment immunoglobulin clonal composition in pediatric B-lymphoblastic leukemia.

  • Read more about Prognostic impact of pretreatment immunoglobulin clonal composition in pediatric B-lymphoblastic leukemia.

Venetoclax and dinaciclib elicit synergistic preclinical efficacy against hypodiploid acute lymphoblastic leukemia.

  • Read more about Venetoclax and dinaciclib elicit synergistic preclinical efficacy against hypodiploid acute lymphoblastic leukemia.

A report from the Leukemia Electronic Abstraction of Records Network on risk of hepatotoxicity during pediatric acute lymphoblastic leukemia treatment.

  • Read more about A report from the Leukemia Electronic Abstraction of Records Network on risk of hepatotoxicity during pediatric acute lymphoblastic leukemia treatment.

Survival and causes of death in 2,033 patients with non-transfusion-dependent β-thalassemia

  • Read more about Survival and causes of death in 2,033 patients with non-transfusion-dependent β-thalassemia

The EBF1-PDGFRB T681I mutation is highly resistant to imatinib and dasatinib in vitro and detectable in clinical samples prior to treatment.

  • Read more about The EBF1-PDGFRB T681I mutation is highly resistant to imatinib and dasatinib in vitro and detectable in clinical samples prior to treatment.

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