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Multiplatform molecular analyses refine classification of gliomas arising in patients with neurofibromatosis type 1.
Subgroup and subtype-specific outcomes in adult medulloblastoma.
Integrated molecular and clinical analysis of low-grade gliomas in children with neurofibromatosis type 1 (NF1).
Pediatric bithalamic gliomas have a distinct epigenetic signature and frequent EGFR exon 20 insertions resulting in potential sensitivity to targeted kinase inhibition.
Gliomas arising in the setting of Li-Fraumeni syndrome stratify into two molecular subgroups with divergent clinicopathologic features.
Recurrent non-canonical histone H3 mutations in spinal cord diffuse gliomas.
Recurrent KBTBD4 small in-frame insertions and absence of DROSHA deletion or DICER1 mutation differentiate pineal parenchymal tumor of intermediate differentiation (PPTID) from pineoblastoma.
The genetic landscape of gliomas arising after therapeutic radiation.
Deep sequencing of WNT-activated medulloblastomas reveals secondary SHH pathway activation.
Activating NRF1-BRAF and ATG7-RAF1 fusions in anaplastic pleomorphic xanthoastrocytoma without BRAF p.V600E mutation.