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To T or not to B: germline RUNX1 mutation preferences in pediatric ALL predisposition.

  • Read more about To T or not to B: germline RUNX1 mutation preferences in pediatric ALL predisposition.

Mass cytometry detects H3.3K27M-specific vaccine responses in diffuse midline glioma.

  • Read more about Mass cytometry detects H3.3K27M-specific vaccine responses in diffuse midline glioma.

Lack of Flvcr2 impairs brain angiogenesis without affecting the blood-brain barrier.

  • Read more about Lack of Flvcr2 impairs brain angiogenesis without affecting the blood-brain barrier.

Bile acid metabolism is altered in multiple sclerosis and supplementation ameliorates neuroinflammation.

  • Read more about Bile acid metabolism is altered in multiple sclerosis and supplementation ameliorates neuroinflammation.

The Mir181ab1 cluster promotes KRAS-driven oncogenesis and progression in lung and pancreas.

  • Read more about The Mir181ab1 cluster promotes KRAS-driven oncogenesis and progression in lung and pancreas.

Glucocorticoids paradoxically facilitate steroid resistance in T cell acute lymphoblastic leukemias and thymocytes.

  • Read more about Glucocorticoids paradoxically facilitate steroid resistance in T cell acute lymphoblastic leukemias and thymocytes.

CD161 contributes to prenatal immune suppression of IFNγ-producing PLZF+ T cells.

  • Read more about CD161 contributes to prenatal immune suppression of IFNγ-producing PLZF+ T cells.

Schwann cell-derived periostin promotes autoimmune peripheral polyneuropathy via macrophage recruitment.

  • Read more about Schwann cell-derived periostin promotes autoimmune peripheral polyneuropathy via macrophage recruitment.

CRISPR-Cas9 screen reveals a MYCN-amplified neuroblastoma dependency on EZH2.

  • Read more about CRISPR-Cas9 screen reveals a MYCN-amplified neuroblastoma dependency on EZH2.

Long telomeres protect against age-dependent cardiac disease caused by NOTCH1 haploinsufficiency.

  • Read more about Long telomeres protect against age-dependent cardiac disease caused by NOTCH1 haploinsufficiency.

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